ABCC1 p.Pro794Asp
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PMID: 19063607
[PubMed]
Qin L et al: "Residues responsible for the asymmetric function of the nucleotide binding domains of multidrug resistance protein 1."
No.
Sentence
Comment
195
Consistent with their effects on VO4-dependent nucleotide trapping by the D793E mutant protein (Figure 5), both the W653Y and P794D second mutations restored the ability of ATP plus VO4 to induce the shift from high- to low-affinity LTC4 binding (Figure 6A).
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ABCC1 p.Pro794Asp 19063607:195:126
status: NEW